By Charles J. Prestigiacomo, Bernard R Bendok
Covering not just the most recent recommendations but in addition the technological know-how and reason at the back of neuroendovascular therapy, this reference displays the present wisdom base of the endovascular surgical neuroradiology subspecialty. It covers all elements of neuroendovascular surgical procedure, equivalent to the technology of vascular biology to the extra complicated medical functions in acute stroke interventions and AVMs. Written through neurologists, neurosurgeons, and neuroradiologists, this well timed textual content presents readers with a radical overview of the entire concerns pertinent to the endovascular remedy of illnesses of the cerebrovascular method, backbone, head, and neck.
- Technique chapters comprise hassle avoidance and management
- High-quality, exact illustrations and updated photographs advisor the reader via medical options and technically hard procedures
- Covers issues which are frequently neglected yet are severe to knowing the dynamics of endovascular therapy, similar to using anticoagulants or procoagulants and the biophysics of vascular disease
- Each bankruptcy ends with a precis which distills and highlights the foremost takeaways for that subject
Endovascular Surgical Neuroradiology is a key source that trainees in addition to extra professional clinicians will confer with time and again over the process their careers.
Read Online or Download Endovascular Surgical Neuroradiology: Theory and Clinical Practice PDF
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Additional info for Endovascular Surgical Neuroradiology: Theory and Clinical Practice
Thrombophilic and hemophilic states can be results of genetic defects, adverse drug effects, and sequelae from disease. This section gives brief overviews of selected pathological states affecting hemostasis. Plasminogen Activator Inhibitor Thrombophilic States Plasminogen activator inhibitor-1 (PAI-1) does exactly what its name suggests. By forming an inert, nonreactive covalent complex with either tPA or uPA, PAI inhibits the activation of plasminogen and the subsequent fibrinolysis. 112 PAI-1 and PAI-2 play the largest roles in fibrinolysis inhibition.
J Biol Chem 2004;279(17):17241–17249 PubMed 38. Komiyama Y, Pedersen AH, Kisiel W. Proteolytic activation of human factors IX and X by recombinant human factor VIIa: effects of calcium, phospholipids, and tissue factor. Biochemistry 1990;29(40):9418–9425 PubMed 2 Hemostasis 39. Lawson JH, Mann KG. Cooperative activation of human factor IX by the human extrinsic pathway of blood coagulation. J Biol Chem 1991;266(17):11317–11327 PubMed 40. van Dieijen G, Tans G, Rosing J, Hemker HC. The role of phospholipid and factor VIIIa in the activation of bovine factor X.
Heparin-induced thrombocytopenia (HIT) often manifests as thromboembolic events despite what its name would lead us to believe. In HIT, an immunoglobulin G (IgG) antibody, directed against heparin bound to platelet factor 4, also binds to platelets via an Fc receptor and thereby activates them. 133 The pathophysiology seems to be related to the ongoing inflammation and release of inflammatory factors that lead to an ongoing hypercoagulable state. 134 The link between cancer, cancer treatment, and thrombosis has long been established and is also dependent on the same principle.